Research Topic Directory Cardiovascular Physiology

Name E-mail Address Phone Department
970-491-4300Biomedical Sciences
970-491-2009Biomedical Sciences
970-491-3605Biomedical Sciences
970-491-2533Biomedical Sciences
970-491-3186Biomedical Sciences

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                    [ProfileText] => Dr. Amberg is Assistant Professor of Cardiovascular Pharmacology.  His laboratory investigates how reactive oxygen species induce pathological changes in arterial smooth muscle associated with hypertension. Results from this research aim to provide insight into understanding why clinical trials with antioxidant supplements generally produce negative results and thereby lead to new pharmacological tools for the clinical prevention and management of hypertension. Blocking the increased calcium channel function should prevent arterial contraction leading to high blood pressure and diseases such as stroke.  Events underlying altered calcium channel function during hypertension are being explored by a combination of total internal reflection fluorescence microscopy, voltage-clamp electrophysiology, fluorescent calcium imaging and video microscopy of intact arteries to seek new therapies for arterial disease.
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                    [ProfileText] => Teaching Interests: cardiovascular and pulmonary physiology and pathophysiology, comparative physiology, and application of physiologic principles to human and veterinary medicine.

Courses: VM 610-611 Foundations of Veterinary Medicine I/II, VM 618 Veterinary Physiology and Histology, VM 618 Functional Anatomy, VM 7222 Veterinary Pharmacology, and BMS 501 Mammalian Physiology II.

Methods: incorporation of demonstrations and clinical footage into the classroom environment, web-based resources and learning, review of current literature, and provision of learning opportunities requiring critical thinking and practical application of principles learned.
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                    [ProfileText] => My research is generally focused on the integrative neuroscience of stress. Specific projects combine behavioral neuroscience, cardiovascular and endocrine physiology, and neurocircuit analysis to determine the mechanistic basis of stress effects on health. Current studies are particularly focused on the neurobiological mechanisms linking depression and cardiovascular disease. Stress-induced changes in cardiovascular reactivity represent a significant detriment to long-term health and mortality. Changes in autonomic regulation following chronic stress may generate a predisposition to co-morbid depression and cardiovascular disease. To identify the neural substrates linking chronic stress, cardiovascular disease, and depression, my laboratory uses a variety of tools including optogenetics, radiotelemetry, immunohistochemistry, connectional anatomy, confocal microscopy, plasma hormone analysis, and social/affective behavioral assays. 
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